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Isolated platelets from humans, dogs, and monkeys produce thromboxane B2 (TXB2) in response to thrombin, arachidonic acid, epinephrine, or ionophore A23187. In this study, we found that the relative level of TXB2 biosynthesis in response to these stimulants is related to the amount of extracellular cyclooxygenase activity present. Platelets isolated from dogs and monkeys produced more TXB2 in response to arachidonic acid and epinephrine than platelets from humans. Incubation of isolated human platelets with the irreversible inhibitor of arachidonate metabolism, mepacrine, inhibited the arachidonate-induced generation of TXB2. Inhibition of thrombin-stimulated TXB2 formation by mepacrine was counteracted by prostaglandin E1. In contrast, cyclooxygenase activity and platelet aggregation in response to arachidonic acid were not inhibited by mepacrine. Activation of platelet phospholipase A2 activity by bovine serum albumin was also diminished by mepacrine, but the inhibitory effect was not reversed by prostaglandin E1. Incubation of platelets with methylarachidonyl
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The complexity of the biofilms and interactions between biofilm and clean surface make it very difficult to remove. Although multiple mechanisms have been used for biofilm removal, the most promising approach is to remove the biofilm from the surface at the early stage. Recently, the “memory effect” of the biofilm has been demonstrated to be an important factor leading to the stability and shear-resistance of the mature biofilm. However, the process of biofilm formation is very complicated; it is difficult to control the formation of early stage biofilms in the process of growing. This can create a situation that the mature biofilm could not be prevented because of the memory effect of the initial biofilm. To solve this problem, this research introduces a modified crystal structure to remove the initial biofilm. As the crystal structures are limited by the size of a periodic cell, different sizes of clusters or particles are attached to different crystal surfaces. Compared with the current state of the art, this method can effectively limit the formation of memory effect in the biofilm process and prevent the formation of mature biofilms. In addition, the method can be applied to various kinds of solid surfaces because it does not need complicated preparation of surface.Q:

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